Volume : 03 Issue : 03
INTERNATIONAL JOURNAL OF DIAGNOSTICS AND RESEARCH [ISSN No.: 2584-2757]
Haemorrhoids (Arsha) [12,13]
Disease progression
:
Clinical Features
tables provide an overview of the application of
Shatkriyakala in four common surgical diseases,
correlating each stage with disease progression,
clinical features, and stage-wise management. This
demonstrates the transition from early reversible
pathology to advanced complications, along with
Stage
(Shatkriyakala)
Management
Predisposing factors such Heaviness, constipation, Nidana Parivarjana (avoid
as chronic constipation, mild discomfort; early causative factors), high-fiber
prolonged straining,
dosha chaya without
diet, hydration, bowel
regulation, lifestyle
correction.
sedentary lifestyle leading clear pathology.
to venous congestion in
hemorrhoidal plexus
Sanchaya
(Kapha-Vata imbalance
due to Viruddhadhyasan,
Vega Dharana, Ati Asana,
Striprasanga).
corresponding treatment preferences.
[9,10,11]
Increased venous pressure Straining during Shamana Chikitsa, bowel
with persistent congestion defecation, discomfort, regulation, stool softeners,
Appendicitis (Undukpuch shoth)
:
and early vascular
dilatation (Vata-Pitta
aggravation). Dosha
prakopa individually or in
combination.
occasional bleeding;
aggravated bowel
symptoms.
sitz bath; early medical
intervention.
Stage
(Shatkriyakala)
Prakopa
Prasara
Disease progression
Clinical Features
Management
Sluggish gut motility,
luminal stasis with mucus Vague abdominal
inspissation and lymphoid discomfort,mild
Dietary modification,
Langhan-Deepana-Pachana-
Anuloman,Nidanparivarjan.
Sanchaya
hyperplasia ( Kapha
accumulation)
anorexia
Progressive dilatation of
hemorrhoidal veins with
early pile formation and
mucosal irritation; doshas doshic spread.
spread via Dhamanis.
Itching, mild bleeding Conservative management—
per rectum, mucus
discharge; systemic
dietary measures, laxatives,
topical therapy; Shodhana
(purificatory measures) if
required.
Low fiber diet, refined
carbohydrate intake, and
irregular bowel habits
contribute to luminal
obstruction (fecolith,
stricture, tumor, or
parasites), resulting in
mucosal irritation and early
inflammation (Vata–Pitta
aggravation).‖
Mild abdominal pain,
bowel
irregularity,localized
mucosal edema and
congestion
Early intervention-Shamana
chikitsa, diet and lifestyle
correction
Prakopa
Localization in Gudavali Poorvarupa: anorexia, Dosha–Dushya targeted
(ano-rectal region) with emaciation, anemia, pain treatment; medical therapy,
formation of hemorrhoidal in anal region; later pain, local applications, and
masses due to tissue
weakness and vascular
engorgement (correlates
with Grade I–II
bleeding, intermittent
prolapse.
procedures like sclerotherapy.
Sthanasamshraya
Obstruction leads to
bacterial proliferation and
release of inflammatory
mediators, causing
increased intraluminal
pressure, edema, and
mucosal ulceration
facilitating bacterial
translocation into the
submucosa and peri-
appendiceal tissues, thereby
initiating an early
hemorrhoids).
Antimicrobial and anti-
Increasing pain, nausea, inflammatory drugs by
Fully manifested
hemorrhoids with
Prolapse, bleeding,
discomfort, mucus
discharge; distinct types hemorrhoidectomy depending
of Arsha with classical on grade; Vyadhi-specific
Kshara karma, rubber band
ligation, or
Prasara
low-grade fever
parental route,IV Fluids,
vitals monitoring.
significant prolapse and
vascular changes (Grade
II–III/IV). Clear disease
expression after Dosha–
Dushya Sammurchana.
Vyakti
Bheda
features.
chikitsa.
inflammatory cascade.
Localized appendiceal
inflammation-mucosal
Vague abdominal
Sthanasamshraya edema, compromised blood discomfort,tenderness, Antibiotics, IV Fluids.
Complicated
Severe pain, non-
Surgical hemorrhoidectomy,
flow, local irritation ( Ama mild fever,malaise
involvement)
Severe Right lower
hemorrhoids—thrombosis, reducible mass, profuse excision of thrombosed piles,
strangulation, ulceration, bleeding; complications emergency care;
irreducible prolapse;
chronic progression and
differentiation.
quadrant pain,
McBurney’s point tre
like Gulma, Plihodara Vyadhipratyanika chikitsa.
etc.
Established acute
appendicitis with clear
clinical manifestation
enderness, vomiting,
nausea with clinical
signs such as-muscle
guarding,rovsings
Appendectomy(lap/open)
with antibiotic coverage.
Vyakti
Bheda
sign,psos sign,obturator
sign,leukocytosis
Fistula-in-ano (Bhagandara) [13,14,15]
:
Complicated
appendicitis—
perforation,gangrene, per-
appendiceal abscess,
peritonitis,
Heamodynamically
unstable, Severe
systemic detoriating
Emergency surgical
intervention-Laparotomy
(emergency open
Stage
(Shatkriyakala)
Disease progression
Clinical Features
Management
signs and septic shock, appendectomy with peritoneal
guarding, rigidity lavage)
sepsis,mucocoele
Predisposing factors such Mild itching, perianal
as poor hygiene, chronic
constipation, and
cryptoglandular stasis
leading to micro-
inflammation in anal
glands (Kapha-Pitta
imbalance).
Hygiene maintenance, dietary
correction (high-fiber diet),
bowel regulation, Nidan
Parivarjan such as avoiding
long term sitting on hard
surfaces-sqatting posture.
discomfort.
Cholecystitis (Pittashaya Shoth) [9,10,11]
Disease Progression (Modern
Correlation)
Sanchaya
Stage (Shatkriyakala)
Clinical Features
Management
Biliary stasis with thickened
bile (sludge formation) due to Dyspepsia, heaviness
Low-fat diet, Deepana-
Pachana, Nidana Parivarjan,
Obstruction and infection Pain, mild swelling,
of anal glands with burning sensation,
bacterial proliferation discomfort during
causing early inflammation defecation.
(Vata-Pitta aggravation).
Pitta-kapha Shamana
Chikitsa, sitz bath, stool
softners, medicated oil basti
chikitsa, antibiotics (if
required).
Sanchaya
impaired gallbladder motility after fatty meals, mild Antacids, lifestyle
abdominal discomfort. modification (avoid heavy
meals)
and bile supersaturation
(Kapha-Pitta accumulation).
Prakopa
Prasara
Aggravation leading to
gallstone formation and/or
intermittent cystic duct
obstruction causing mucosal
irritation (Pitta-Kapha
aggravation).
Fat intolerance,
Spread of infection into
surrounding tissues
(intersphincteric space)
Severe throbbing pain, Antibiotics, anti-inflammatory
swelling, redness and
discomfort at perianal
bloating, mild right
hypochondriac
discomfort (biliary
colic).
Pitta-Kapha shamana, dietary
regulation, analgesics(except
opiods)observation.
medications, local
fomentation, incision and
drainage (I&D).
Prakopa
Prasara
leading to anorectal abscess region, fever.
formation.
Persistent obstruction leads to
inflammatory changes in
gallbladder wall with edema,
chemical inflammation, and
early infection.
Medical management—
analgesics(opiods are
contraindicated), anti-
inflammatory drugs,
antibiotics, monitoring.
Localization of abscess
(Guda Vidradhi) with pus erythema, localized
collection and tissue pain, possible
Tender swelling,
Incision and drainage, wound
care,kshar-application for
early sinus tract,
Increasing right upper
quadrant pain, nausea,
vomiting, low-grade
fever.
Sthanasamshraya
damage; early sinus tract discharge,difficulty in antibiotics,regular bowel
formation may begin.
sitting and defeacation. habits, monitoring for tract
formation.
Localization of inflammation
in gallbladder—acute
Sthanasamshraya cholecystitis with cystic duct
obstruction, gallbladder
distension, and wall thickening.
Right upper quadrant
Formation of a well-
defined fistulous tract
connecting anal canal to
perianal skin (post-abscess in perianal
stage).
Persistent pus discharge Ksharasutra therapy,
from external opening, fistulotomy/fistulectomy, or
recurrent pain,induration seton placement.
pain, positive Murphy’s Hospitalization, IV fluids,
sign, Boa’s sign, fever, antibiotics, supportive care.
vomiting.
Vyakti
region,swelling,itching.
Established acute cholecystitis Severe RUQ pain, fever,
Complex fistula with Chronic discharge,
multiple branching tracts, multiple openings,
Advanced surgical
management (fistulectomy,
advancement flap),
Ksharasutra in complex
cases, long-term follow-up.
with marked inflammation,
leukocytosis, guarding, Definitive management—
positive imaging laparoscopic cholecystectomy
confirmation (USG showing findings, jaundice, clay with antibiotics,ERCP,MRCP.
Vyakti
Bheda
leukocytosis, and imaging
recurrence, fibrosis, and
possible sphincter
induration, recurrent
abscess,
wall thickening, stones).
colored stool
involvement leading to
incontinence risk. Chronic
variants described by
acharya sushruta-
shatponak, paristravi,
upnaha,unmargi,etc
Bheda
Complicated cholecystitis—
gangrene, perforation,
empyema of gallbladder,
peritonitis,pancreatitis, sepsis.
Severe systemic illness, Emergency surgical
high fever, rigidity,
signs of sepsis.
intervention, drainage,
intensive care management.
Copyright @ : - Dr.Batra Khushbu Inter. J.Digno. and Research IJDRMSID0111 |ISSN :2584-2757
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